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Our findings provide valuable insights into how reprogrammed glucose metabolism drives HCC progression, identifying key metabolic activities that could be leveraged for therapeutic targeting, and offering potential strategies for improving HCC treatment

Bongiorno R, Colombo MP, Lecis D

We find that each phospho-null mutation within the GAP-F5 protein significantly yet partially reduces G2019S LRRK2-mediated phosphorylation relative to WT GAP-F5, however, none of these three residues serve as a dominant phospho-site as they have equivalent effects ( Fig

K.FoktI.et al (2009)

1 Identification of candidate gene of qCR2 locus

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