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Heme oxygenase-1 reduces cellular and mitochondrial ROS production in COPD fibroblasts, which in turn improves mitochondrial respiration, glycolysis and ATP recovery ( Lipid metabolism in myofibroblasts Lipid metabolism may affect PF via several pathways, including phospholipid synthesis, lipid peroxidation products, and lipid mediators

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However, the available evidence strongly suggests that smoking acts as an independent risk factor that exacerbates the severity and progression of chronic viral hepatitis, increasing the risk of advanced fibrosis and cirrhosis, and potentially impacting treatment efficacy

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NADPH is involved in the cycling of the GPX4 antioxidant system, and its depletion limits the antioxidant function of GSH-GPX4, inducing ferroptosis [56, 57]