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Despite the decrease in ACE2 levels, L-carnitine supplementation did not increase the ACE1/ACE2 ratio, which remained low compared to the control group, suggesting the prevention of a spike in inflammation

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Furthermore, measurement of enzyme activities showed decreases in complexes III [16] and IV [15] and no change in complexes I and II

Insulin released after eating a carbohydrate may improve the uptake of L-carnitine into muscle cells

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Also, the free passage of acetyl-CoA through the nuclear pore complex facilitates the traffic of cytosolic acetyl-CoA to the nucleus [142]