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In addition, this review discusses the multilevel pathophysiological mechanisms involved in acute pancreatitis, including aberrant enzymatic activation, calcium overload, impaired autophagy, inflammatory responses, and various forms of pancreatic acinar cell death

Keywords: post-acute sequelae of SARS-CoV-2, COVID-19, Long COVID, inflammation, metabolism, immunosenescence INTRODUCTION Long COVID (LC) is a multisystem condition following the acute phase of COVID-19, in which individuals experience persistent or new COVID-attributed symptoms including fatigue, cognitive impairment and post-exertional malaise (1, 2)

Snchez-Redondo IR, Alejo LB, Revuelta C, De Pablos R, Ibaez M, Prez-Lpez A, Lucia A, Barranco-Gil D, Valenzuela PL

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Effects of excision repair cross-complementation group 1 (ERCC1) single nucleotide polymorphisms on the prognosis of non-small cell lung cancer patients

Through the RNA sequencing analysis of mice exposed to narrow band noise and treated with MSCs, the marked upregulation of genes related to immune modulation, hypoxia response and mitochondrial function was observed, alongside the downregulation of genes associated with synaptic remodeling and calcium homeostasis