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This can promote further inflammation and excessive levels of pro-inflammatory mediators
Increased platelet aggregation and in vivo platelet activation after granulocyte colony-stimulating factor administration
However, our findings revealed that analogs with a single deletion of His or Trp were not highly effective as antagonists
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gallinarum ) contribute to SLE development by impairing intestinal barrier function, activating autoimmune responses, and sustaining systemic inflammation (Fig
