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showed that miR-221 significantly suppressed the proliferative ability of both DPCs and DSCs, while no inhibitory effects were observed for miR-125b, miR-106a, or miR-410 on the proliferation of DPCs and DSCs (Fig

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The use of JHU08, a prodrug version of the glutamine antagonist DON, which becomes active within the TME, circumvents several challenges faced by metabolic immunotherapy

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