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doi: 10.1016/S1359-6446(03)02703-X

It addresses one specific hormonal mechanism but does not stimulate follicles directly or address inflammation

One possible explanation for this relationship is that oxidants generated by phagocytosing macrophages may diffuse into the extracellular medium, where they pose an oxidative threat to G6PD-deficient erythrocytes

These results suggest that the aberrant regulation of G6PD signaling and/or metabolic pathways may be associated with the pathogenesis of these diseases

Acknowledgments The authors sincerely thank the Department of Product Evaluation (Shanghai Jiyan Biomedical Development Co., Ltd.) for their technical support in skin experiments

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