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Overall, mounting evidence suggests that targeting CD38 and related NAD + -consuming enzymes, such as PARPs, has great potential as an NAD + -boosting target to extend human healthspan, and several pharmacological approaches are in development to inhibit CD38 and its NADase activity 232 (TABLE 1)
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The Lancet
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The N-terminal Tyrosine protects against enzymatic degradation by aminopeptidases
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