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Retrieved 13 July 2026.

Under these pathological conditions, vitamin D regulates immune function by inhibiting the activation of T helper 1 and T helper 17 cells, while promoting the differentiation of Tregs

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(91) discovered that D3R can significantly reduce cell apoptosis induced by tert-Butyl hydroperoxide (t-BHP), protecting MC3T3-E1 cells from oxidative stress damage

p = 0.001) by the IVW-multiplicative random effect model