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These studies suggest that ammonium accumulation may induce programmed cell death of immune cells within TME, such as NK cells, through oxidative imbalance and mitochondria-mediated autophagy, thereby promoting immune evasion by prostate cancer cells

These systems tend to repair oxidative damage as they act as oxidative sensors in signal transduction pathway [4]

This response has been associated with exposure to ultraviolet A (UVA), while ultraviolet B (UVB) does not appear to have a similar effect

E.et al (2019b)

SS-31 stabilizes the inner mitochondrial membrane, protecting neurons from oxidative damage and energy depletion