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The neurogenetic syndrome associated with ADNP mutation, HVDAS, accounts for 0.17% of ASD cases and is therefore considered a high-risk gene [292]

One GSTZ1 encoded protein lacks the glutathione binding site and is, therefore, unlikely to exhibit the enzymatic activity of the other three isoforms

The cell viability, apoptosis and glutathione reductase activity were measured to confirm the development of riboflavin deficiency

Notably, emerging evidence suggests a potential link between folate pathway polymorphisms and autism spectrum disorder ( ASD )

Obstet Gynecol Sci
