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The collective genomics of detoxification enzymes dictates the clinical phenotype in response to AFs exposure

Representative examples and advances of research progress are described below

Cell 169 , 570586 (2017)

Additionally, hypoxia causes Unc-51-like autophagy activating kinase 1 (ULK1) to translocate to the mitochondria and engage with FUNDC1, phosphorylating Ser17 and enhancing the interaction between FUNDC1 and LC3 [108]

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