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doi: 10.1016/j.cotox.2017.11.002 240 GroempingYRittingerK
Thus, mitochondrial dysfunction and oxidative stress can be considered as potential pathogenetic targets for therapeutic correction of the disease by using short peptides
It is considered to be the final step in eukaryotic nitrogen excretion and very important in recycling oxidised NAD +
less likely to require treatment for gout flares, fewer adverse effects due to reduced exposure to NSAIDs
This sequence has a very high affinity for Copper (II) ions, allowing it to chelate (grab onto) copper from the surrounding environment [2]
