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It also promotes an NMDA-receptor-mediated decrease in the number of neurons (29)

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The second mechanism of the toxic action is based on the displacement of biogenic metals from metal-containing complexes

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Mechanistically, augmented ischemia-reperfusion injury in the absence of GPx-1 may be caused by increased NFB activation, as pyrrolidine dithiocarbamate (PDTC), an NFB inhibitor, partially protects against neurotoxicity in this model (82)