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Inflammatory cytokine concentrations are acutely increased by hyperglycemia in humans: role of oxidative stress

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Recent studies have also shown that accumulated uremic toxins can activate AhR during CKD progression, further inhibiting mitochondrial biogenesis by promoting the ubiquitination and proteasomal degradation of PGC1-, thus accelerating renal aging and fibrosis [308]

doi: 10.1089/cap.2007.0066

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