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[DOI] [PMC free article] [PubMed] [Google Scholar] 55.Salisbury BH, Leslie SW, Tadi P

This chemical binding process transforms the fat-soluble toxins into water-soluble compounds

3i) and rescued the anti-proliferative effects of mitochondrial GSH depletion in SLC25A39/40 double knockout cells (Fig

Both Nrf1 and Nrf2 overexpression induced the human GS promoter activity [144]

In the endoplasmic reticulum, it also acts like a built-in quality-control partner, supporting proper protein folding and helping prevent misfolded proteins from accumulating

927 These results suggest that HDAC3 maintains iron homeostasis through the Hippo/YAP pathway and might represent a target to reduce ferroptosis in iron overload-related diseases
