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The therapeutic activity of NAC relies on its ability to provide reducing sulfhydryl moieties and to act as a precursor of GSH to replenish the intracellular GSH pool when this has been depleted in conditions of oxidative stress, in processes of drug detoxification, or in other conditions leading to GSH deficit

In the case of the JNK inhibitor SP600125, which is only soluble in DMSO and requires early treatment, 70% of the protection against APAP toxicity was caused by DMSO and 30% by SP600125 [36]

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However, diabetes driven effects on heart should not be limited on myocardium only

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