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In these studies, autophagy activation was able to attenuate FIN56 or erastin-induced ferroptosis, even in the absence of ROS generation

10.1093/chromsci/bmw060 100 SiesH.BelousovV

Studies have explored growth hormone receptor upregulation and VEGF pathway activation as potential mechanisms that could explain this multi-tissue activity though the complete mechanistic picture remains an active area of investigation

Thus, GPX4 inhibitors could induce ferroptosis, and targeting GPX4 might be a feasible therapeutic strategy

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SURMOUNT arthralgia rates match placebo