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Antisense therapy in a rat model of Alexander disease reverses GFAP pathology, white matter deficits, and motor impairment

Pathobiology of Hemophilic Synovitis I: Overexpression of Mdm2 Oncogene

By transferring mitochondria from Parkinsons patients into mtDNA-depleted cells that were otherwise healthy, researchers identified defects in the electron transport chain and heightened oxidative stress, underscoring the role of mtDNA alterations in Parkinsons disease (Miller et al., 1996

Leukocyte telomere length is reduced in patients with major depressive disorder

This highly flexible arrangement facilitates simultaneous binding of several host endocytic proteins and modulation of the hosts PM (Fig

Hayes and Pulford, 1995)
