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Additionally, in the acute lung injury model, significantly upregulating the expression of FXR can inhibit the phosphorylation of p38 MAPK and NF-B p65, reduce the release of inflammatory factors, and thereby protect lung tissue (108)

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It serves as an obligate co-substrate for multiple enzyme families, including glutathione peroxidases (GPx), glutathione S-transferases (GST), and glutaredoxins, where it contributes to redox-regulated biochemical pathways and enzyme-mediated signaling processes

These findings highlight the urgent need for future therapies that can simultaneously improve glycemic control and restore normal platelet function in diabetic populations

2: You must maintain a good skincare routine