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It has been proposed that the pathophysiology of IIM could be attributed to increased cell death, inadequate removal of cell debris, and excessive autoantigens and pro-inflammatory cytokines, which eventually result in over-stimulated immunological and inflammatory responses (152)

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[DOI] [PubMed] [Google Scholar] Jean P.A., Bailie M.B., Roth R.A

In HCC tissue, immunohistochemical analyses detected GPx2 overexpression when compared to adjacent non-tumor tissues [313]

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