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As expected, silencing of endogenous KEAP1 increased luciferase activity driven by ARE elements under Cyss depletion conditions, whereas simultaneous expression of exogenous shRNA-resistant KEAP1 restored basal expression (Fig

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Aberrant hydroxymethylation of ANGPTL4 is associated with selective intrauterine growth restriction in monochorionic twin pregnancies

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Overexpression of ACSL4 in a GBM cell line was shown to downregulate GPX4 while increasing the expression of relevant ferroptosis markers [86]

Association of growth differentiation factor 15 (Gdf15) polymorphisms with Serum Gdf15 and ferritin levels in -thalassemia