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In addition, extracellular lactate stabilizes HIF-2, and HIF-2 then transactivates MYC, increasing the levels of glutamine transporters and GLS1, in turn resulting in increased glutamine catabolism 72

doi: 10.1007/s10571-023-01405-w, 74 GuzelT

& Rong, P.-J

While many people obtain adequate B12 from their diet, others may require additional support due to deficiencies or specific health needs

Our results thus indicated that AITC treatment led to glutathione depletion which contributed to the higher susceptibility of glutathione mutants, but the molecular mechanism behind this physiological response is likely more complicated

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