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To identify the molecular changes that result in elevated striatal dopamine levels, the group analysed the transcriptome of the impacted neurons using RNA sequencing from our previously optimised approach

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To address this, they applied a regression-based method (RePACT), which revealed a diabetes-associated epigenetic signature: decreased chromatin accessibility at motifs for HNF1A/B and RFX6, and increased accessibility at motifs for NEUROD1, NFY, and TP53