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The proposed therapeutic mechanism involves the down-regulation of c-Jun-N-terminal kinase (JNK) by inducing Gadd45-beta activity, which ultimately disrupts a mechanism of mitochondrial damage

Key Takeaway: Glutathione supports joint health by acting as a biological shield that neutralizes reactive oxygen species and helps regulate the signaling proteins responsible for the body's inflammatory response

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For tissue homogenates, 5 g total protein were used and the results normalized to amount of total input protein and expressed as ng GPX3/g protein

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