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HER2 gene amplification and EGFR expression in a large cohort of surgically staged patients with nonendometrioid (type II) endometrial cancer

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Role of IGF-1 in renal ischemia Post-ischemic regeneration leads to increased renal IGF-1 expression, promoting proliferation of tubular and non-tubular cells at the kidney regenerative zones by binding IGF1R.32 Studies using animal models of ischemic acute renal injury have shown that IGF-1 administration is useful to reduce renal protein catabolism, resulting in enhanced renal cell proliferation, accelerated recovery of renal function, and improved kidney histology.33,34 These regenerative effects after ischemia-induced acute tubular necrosis are also achieved by exogenous administration of recombinant IGF-1, resulting in a lower mortality in comparison with animals treated with vehicle or GH.34,35 However, despite these promising results in animal models, administration of recombinant IGF-1 (100g/12h

Lyme disease is caused by the bacteria Borrelia burgdorferi, which is spread by ticks

We found that AMPH can cause endocytosis of NET as well as EAAT3 in NE neurons

10.1073/pnas.171178698 55 Shetab-BoushehriS