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Notably, the endogenous antimicrobial peptide Beta-defensin-2 is upregulated following I/R injury, and moderate suppression of this upregulation may attenuate lung damage by inhibiting excessive inflammation and oxidative stress[92, 93], highlighting its complex role in injury pathogenesis

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It should subside on its own

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[DOI] [PubMed] [Google Scholar] Vlaming MLH, Mohrmann K, Wagenaar E, de Waart DR, Oude Elferink RPJ, Lagas JS, van Telligen O, Vainchtein LD, Rosing H, Beijnen JH, Schellens JHM, Schinkel AH

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