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In addition, we discuss the hijacking of the iron regulatoy pathway and the antioxidant response, both of which are typical in viral infections

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The last term is the activation of CBS by oxidative stress [25, 26], which is represented by the concentration of H 2 O 2

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EG-treated rats exhibited severe oxidative stress-induced damage, as evidenced by significantly reduced antioxidant enzyme activities (SOD, CAT, and GSH) and elevated GPx activity

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