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Depletion of bioavailable nitric oxide in the vasculature by excess oxidants causes endothelial dysfunction, which is characterized by a decrease in endothelium-dependent vasodilation while retaining the ability of vascular smooth muscle cells to vasodilate in response to nitric oxide donors, such as sodium nitroprusside [53]

doi: 10.1111/j.1399-0004.2011.01728.x

41 When T cells are deprived of Trp and Arg, activated T cells cannot enter the S phase, which proves that Trp and Arg are key substances for T cells to enter the cell cycle

The relationship between lipoprotein(a) and risk of cardiovascular disease: a mendelian randomization analysis

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Evaluation of nebulised hypertonic saline (7%) as an adjunct to physiotherapy in patients with stable bronchiectasis