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This type of cell death differs from conventional forms, such as autophagy, apoptosis, and necrosis, as it is primarily triggered by the accumulation of lipid peroxides

IAC, the reaction product of TEMPO and presumably also TEMPOL, showed protective effects in animal models of various diseases, including colitis, Alzheimers disease, and diabetes (Novelli et al., 2007

One of these by-products is malondialdehyde (MDA)

Both priming and activation are blocked by treatment with genistein, a tyrosine kinase inhibitor, suggesting an important role for tyrosine kinase signaling pathway in activating NADPH oxidase (McLeish et al., 1998

Osborne, J

[DOI] [PMC free article] [PubMed] [Google Scholar] 43.Begley D.J., Brightman M.W