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Description
When disc material herniates, it triggers an inflammatory cascade involving cytokines, prostaglandins, and other chemical mediators
This in turn may exhibit positive action on wound healing processes
Post-translational regulation of NNMT (e.g., phosphorylation, succinylation) adds complexity and may be targeted in future therapies
If IGF-1 rises meaningfully at follow-up, the clinician needs to suspect batch-quality issues (GH contamination in the compounded product) or an alternate mechanism
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