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After activation in cancer cells, the glycosidic bond is broken, releasing DOX for colon cancer therapy with a fluorescence turn-on response, enabling monitoring of drug localization and action site (Figure 4A)

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BPC-157 Body Protective Compound-157, a pentadecapeptide derived from human gastric juice proteins

Hu J, Hwang SS, Liesa M et al (2012) Antitelomerase therapy provokes ALT and mitochondrial adaptive mechanisms in cancer

Among them, enhanced metabolic detoxification and target site insensitivity are the most common mechanisms [5, 33-35]

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