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Lipolytic effects occur through cAMP-mediated triglyceride hydrolysis in fat cells, while osteoblast activation requires MAPK signaling through growth hormone receptors in skeletal tissue
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Neither ratio has been validated in a human blend trial
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Activation of KEAP1/NRF2 stress signaling involved in the molecular basis of hemin-induced cytotoxicity in human pro-erythroid K562 cells
