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Thus, plasma GFAP levels at post-acute phase (day 14) support the accelerated healing of CAQK indicated by the neuroinflammatory markers examined above

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Serum albumin levels appear to be a useful adjunct in evaluating the risk of toxicity levels because albumin binds bilirubin in a ratio of 1:1 at the primary high-affinity binding site (Hansen & Aslam, 2017)

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CLOCK and BMAL1/ARNTL disruption is implicated in inflammation, mitochondrial dysfunction, dopamine, and serotonin disturbance in BD patients [36, 168], as well as in iron dysregulation [169]