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In this review, we summarize the latest findings on glucose metabolism in PD and its clinical relevance, describe the underlying mechanisms by which abnormal glucose metabolism may contribute to PD, and outline potential therapeutic drugs that target glucose metabolism

Concordantly, a 24-h acivicin treatment of tbb-6::GFP worms increased GFP fluorescence by approximately 6-fold (Fig

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Best practices usually include: Start with the lowest dose in the dosage range

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