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Accordingly, there is a serious need for new therapies to treat CRC and to overcome this resistance

For GLR3.5 mutants, Glu treatment had no effect on stomata closure, further supporting the signaling function of Glu by activating GLRs-mediated Ca 2+ signaling pathway (Yoshida et al., 2016)

doi: 10.1186/s12986-022-00696-y 96

In vitro, astrocytes derived from induced pluripotent stem cells obtained from patients with FTD-MAPT showed hypertrophy, elevated GFAP protein levels, disease-associated changes in TAU expression, increased vulnerability to oxidative stress, and protein ubiquitination 231

Like HO-1, other Nrf2-regulated antioxidant genes such as glutamate-cysteine ligase, modifier subunit (Gclm), glutamate-cysteine ligase, catalytic subunit (Gclc), peroxiredoxin 2, glutathione peroxidase, have been shown to protect against atherosclerosis

252 Several studies affirm aberrant activation of this pathway and its promotion of malignant phenotypes in HCC via different mechanisms