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Resistin decreases expression of endothelial nitric oxide synthase through oxidative stress in human coronary artery endothelial cells
Oxidative stress is of great importance in the pathogenesis of HE, while the underlying mechanism remains to be clarified

In a sense, it is the supervisor of your body's cellular defence department

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The nasal mucosa is packed with blood vessels
These data implicate the PIF4 N-terminal IDR in recruiting cofactors for promoting thermomorphogenesis, while the HLH module is necessary but only needed for its dimerization/oligomerization function
