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While there is increasing evidence supporting the neuroprotective properties of UA in Parkinsons disease and Alzheimers disease, gaps in knowledge still exist regarding the underlying mechanisms and how to effectively translate these benefits into clinical practice

Inhibition of IL-17 could ameliorate A-induced neurotoxicity and cognitive decline in C57BL/6 mice by downregulating the TRAF6/NF-B pathway, which provides new clues for the mechanism of A-induced cognitive impairments.

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Here, we review mitochondrial iron utilization and its intersection with disease.