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Laboratory testing cannot directly measure endothelial function, arterial elasticity, or plaque burden, but it can identify many of the lipid, inflammatory, metabolic, and kidney-related forces that accelerate vascular damage

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Ischemic preconditioning reduces endoplasmic reticulum stress and upregulates hypoxia inducible factor-1 in ischemic kidney: the role of nitric oxide

The rationale for studying them together is straightforward: if two compounds affect different but converging pathways involved in tissue repair, their combined application in experimental models may reveal interactions that cannot be observed when studying either compound in isolation