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Botulinum neurotoxin type A (BoNT/A), an exotoxin from Clostridium botulinum, reversed the downregulation of type II collagen and the upregulation of MMP-13 induced by LPS in chondrocytes, alleviating LPS-induced chondrocyte toxicity, activating the SLC7A11/GPX4 antiferroptotic system, and enhancing mitochondrial function

BasuRay, S., Smagris, E., Cohen, J

2024 Jul 13];345(3):473-82

While female patients demonstrated a higher burden of methylation dysregulation, they may possess certain biological mechanisms that mitigate more severe disease progression, potentially explaining the sex-specific differences in schizophrenia susceptibility (Zhou et al., 2023

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NSS mitigated MSG-induced renal impairments by ameliorating oxidative stress and exerting anti-apoptotic effect