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Up to now, two different mechanisms can be considered to operate in the anticancer activity of Casiopeinas

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Following a single mild TBI in mice, glucose utilization in the hippocampus and sensorimotor cortex increased in the first 3 days following injury, while rTBI (a second injury 3 days following the first injury) failed to elicit the same immediate response (63)

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This cascade of oxidative damage and barrier dysfunction is increasingly recognized as a core mechanism linking neuroinflammation to the neurodevelopmental abnormalities observed in ASD (Russo et al., 2018)

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Antioxidant therapeutics in Parkinsons disease: current challenges and opportunities

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