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Disulfiram-Cu 2+ complexes inhibit ubiquitinated protein degradation through ATP-synthase-dependent ubiquitination pathways, induce mitochondrial metabolic dysfunction, acute proteotoxic stress, and ultimately cause cuproptosis ( 2.3 Cu excretion and lysosomal autophagy Lysosomal autophagy, a protective mechanism, clears abnormal proteins and damaged organelles through lysosomes

[PMC free article] [PubMed] [Google Scholar] Guevara, B

Differential sensitivity of inflammatory macrophages and alternatively activated macrophages to ferroptosis

Mak TW, Grusdat M, et al

Involvement of probiotics and postbiotics in the immune system modulation

Available from: /pmc/articles/PMC6359489/