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These vasodilatory mechanisms are diverse but share two distinct characteristics in that the activity of each of the differing mechanisms increases in direct response to increasing intensities of exercise and those mechanisms all initiate the synthesis of nitric oxide (NO)

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An early study showed that 250 M acetyl-l-carnitine did not affect axon regeneration but significantly reduced the rate of death of neurons from the DRG of aging rats, suggesting its neuroprotective action [14]

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These metabolic shifts may contribute to improved endurance capacity and delayed fatigue onset during prolonged physical activity