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Description
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Participants The study was conducted between March 2011 and June 2012 in the Child Development and Behavior Research Center of Harbin Medical University, Harbin, China
The ability to cross the bloodbrain barrier is a key feature explaining the tropism of these compounds to the central nervous system where they accumulate and once a threshold is reached, toxicity will ensue, characterized by motor and cognitive function deficits which are especially pernicious during in utero development
This antioxidant has been associated with a delay in the clinical onset of the disease (Table 1) in transgenic mice that express mutant copies of the gene encoding SOD1 (an animal model of ALS) (Gurney et al., 1996)
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Proven Benefits: Studies show it reduces oxidative stress and enhances glutathione levels
