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Autophagic flux tracing using tandem mRFP-GFP-LC3 probe showed that increase of autophagic flux caused by GSH depletion is augmented by rapamycin and attenuated by autophagic inhibitors 3-MA and Baf-A1 (Fig
Some GSTs display glutathione peroxidase activity and these GSTs can detoxify toxic lipid hydroperoxides that accumulate during infections
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Protective effect of nitric oxide on ischemic retina
