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Nicotinamide and EGCG reduce A accumulation and protein oxidation in aged 3xTg-AD hippocampal neurons Since intracellular A aggregates increase across the lifespan of the 3xTg-AD model in vivo [38], and in vitro [1] and a hallmark for brain aging and the progression of AD is protein oxidation, including tyrosine nitration [1, 39], we immunostained neurons for A accumulation and protein nitration and evaluated the effects of treatment with nicotinamide and EGCG

In addition, the size of the study cohorts was frequently limited, and they were usually from Western countries 4,6,7

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