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The Lancet Diabetes & Endocrinology , 10(1), 45-57

These results are consistent with the notion that lipid peroxidation, including that initiated by A42-resident sulfuranyl free radicals, is a key step in formation of HNE and other products of lipid peroxidation and consequent neuronal death in AD and more broadly in the pathogenesis and progression of AD

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Abstract Main conclusion Reactive nitrogen species mitigate the deteriorative effect of accelerated seed ageing by affecting the glutathione concentration and activities of GR and GPX-like