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Mild renal impairment can be repaired by the kidneys themselves, while severe AKI injury can result in atypical repair, leading to CKD or even ESRD [8]

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These limits are well below the physiological concentrations of these metabolites in whole blood (Fig

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Overall, the discrepancy in AIBL-pK a trends with substituent type (Supplementary Note 2) suggests that, in the search for a bond that has a relationship with pK a over a wide variety of substituent patterns/types, it is logical to look to the enolic hydroxyl group, i.e