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and glutaredoxins, which use GSH to repair oxidised protein thiols

[DOI] [PubMed] [Google Scholar] 411.Kahn V

No validated biomarkers currently exist to confirm neuroprotective effects in patients, and thus, therapeutic efficacy is often inferred from cognitive test scores rather than biological indicators of disease modification (see [567])

Blocking NNMT preserves the NAD+ precursor pool (1.2-1.6x increase in intracellular NAD+), increases SAM availability for epigenetic methylation, and activates SIRT1/SIRT3 signaling

Glutaredoxin exerts an antiapoptotic effect by regulating the redox state of Akt

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