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Preclinical research from Monash University established that HGH Fragment 176-191 stimulates lipolysis and inhibits lipogenesis in adipocyte models at concentrations where full GH would also produce IGF-1-mediated growth effects

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for MetS and T2D), and because exercise is one of the most potent IGF-1 synthesis/freeing mechanisms, it seems logical to correlate them and establish IGF-1 as a target for future options in the multifactorial treatment for metabolic syndrome